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Cbl家族蛋白介导主要组织相容性复合体依赖性CD4+和CD8+T细胞的发育

Establishment of the major compatibility complex-dependent development of CD4+ and CD8+ T cells by the Cbl family proteins.

作者信息

Huang Fang, Kitaura Yasuyuki, Jang Ihnkyung, Naramura Mayumi, Kole Hemanta H, Liu Liping, Qin Haiyan, Schlissel Mark S, Gu Hua

机构信息

Department of Microbiology, Columbia University College of Physicians and Surgeons, 701 West 168th Street, New York, New York 10032, USA.

出版信息

Immunity. 2006 Oct;25(4):571-81. doi: 10.1016/j.immuni.2006.08.021.

Abstract

Casitas B cell lymphoma (Cbl) proteins are negative regulators for T cell antigen receptor (TCR) signaling. Their role in thymocyte development remains unclear. Here we show that simultaneous inactivation of c-Cbl and Cbl-b in thymocytes enhanced thymic negative selection and altered the ratio of CD4(+) and CD8(+) T cells. Strikingly, the mutant thymocytes developed into CD4(+)- and CD8(+)-lineage T cells independent of the major histocompatibility complex (MHC), indicating that the CD4(+)- and CD8(+)-lineage development programs are constitutively active in the absence of c-Cbl and Cbl-b. The mutant double-positive (DP) thymocytes exhibited spontaneous hyperactivation of nuclear factor-kappa B (NF-kappaB). Additionally, they failed to downregulate the pre-TCR and pre-TCR signaling. Thus, our data indicate that Cbl proteins play a critical role in establishing the MHC-dependent CD4(+) and CD8(+) T cell development programs. They likely do so by suppressing MHC-independent NF-kappaB activation, possibly through downmodulating pre-TCR signaling in DP thymocytes.

摘要

Casitas B细胞淋巴瘤(Cbl)蛋白是T细胞抗原受体(TCR)信号传导的负调节因子。它们在胸腺细胞发育中的作用尚不清楚。在此我们表明,胸腺细胞中c-Cbl和Cbl-b的同时失活增强了胸腺阴性选择,并改变了CD4(+)和CD8(+) T细胞的比例。引人注目的是,突变的胸腺细胞发育为独立于主要组织相容性复合体(MHC)的CD4(+)和CD8(+)谱系T细胞,这表明在缺乏c-Cbl和Cbl-b的情况下,CD4(+)和CD8(+)谱系发育程序具有组成性活性。突变的双阳性(DP)胸腺细胞表现出核因子-κB(NF-κB)的自发过度激活。此外,它们未能下调前TCR和前TCR信号传导。因此,我们的数据表明,Cbl蛋白在建立MHC依赖性CD4(+)和CD8(+) T细胞发育程序中起关键作用。它们可能通过抑制MHC非依赖性NF-κB激活来实现这一点,可能是通过下调DP胸腺细胞中的前TCR信号传导。

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