Department of Psychology, Bar-Ilan University, Ramat-Gan, Israel.
The Gonda Brain Sciences Center, Bar-Ilan University, Ramat-Gan, Israel.
J Child Psychol Psychiatry. 2016 Oct;57(10):1183-93. doi: 10.1111/jcpp.12622. Epub 2016 Aug 30.
Although the effects of early-onset trauma on susceptibility to psychopathology are well-acknowledged, no study to date has followed risk and resilience trajectories in war-exposed young children over lengthy periods and charted predictors of individual pathways.
In this prospective longitudinal study, we followed 232 children, including 148 exposed to repeated wartime trauma and 84 controls, at three time points: early childhood (1.5-5 years), middle childhood (5-8 years), and late childhood (9-11 years). Children were diagnosed at each time point and four trajectories defined: children exhibiting no pathology at any time point, those displaying early pathology that later remitted, those showing initial resilience followed by late pathology, and children presenting chronic pathology across the entire first decade. Maternal behavioral containment during trauma evocation and child social engagement during free play were observed in early childhood and maternal emotional distress self-reported across time.
War-exposed children showed significantly higher rates of psychopathology, with 81% exhibiting pathology at some point during childhood. In middle childhood, exposed children displayed more posttraumatic stress disorders (PTSD), anxiety disorders, and attention-deficit/hyperactivity disorders (ADHD), and in late childhood more PTSD, conduct/oppositional defiant disorders, and ADHD. War-exposed children had more comorbid psychopathologies and number of comorbidities increased with age. Notably, war-exposure increased prevalence of chronic pathology by 24-fold. Maternal factors, including mother's uncontained style and emotional distress, increased risk for early and chronic psychopathology, whereas reduced child social engagement augmented risk for late pathology.
Early-onset chronic stress does not heal naturally, and its effects appear to exacerbate over time, with trauma-exposed children presenting a more comorbid, chronic, and externalizing profile as they grow older. Our findings demonstrate that responses to trauma are dynamic and variable and pinpoint age-specific effects of maternal and child factors on risk and resilience trajectories. Results highlight the importance of conducting long-term follow-up studies and constructing individually tailored early interventions following trauma exposure.
尽管早期创伤对精神病理学易感性的影响已得到充分认识,但迄今为止,尚无研究在较长时间内跟踪观察暴露于战争环境中的幼儿的风险和恢复轨迹,并绘制个体途径的预测因素。
在这项前瞻性纵向研究中,我们在三个时间点跟踪了 232 名儿童,包括 148 名暴露于反复战争创伤的儿童和 84 名对照组儿童:幼儿期(1.5-5 岁)、儿童中期(5-8 岁)和儿童晚期(9-11 岁)。每个时间点都对儿童进行诊断,并定义了四种轨迹:任何时间点均无病理表现的儿童、早期病理表现但后来缓解的儿童、最初有恢复力但后来出现晚期病理表现的儿童、以及整个第一个十年期间均有持续病理表现的儿童。在幼儿期观察到创伤诱发时母亲的行为控制以及自由游戏时儿童的社会参与情况,并在整个过程中自我报告母亲的情绪困扰。
暴露于战争的儿童表现出更高的精神病理学发生率,其中 81%的儿童在儿童期的某个时间点出现了病理表现。在儿童中期,暴露于战争的儿童表现出更多的创伤后应激障碍(PTSD)、焦虑障碍和注意缺陷/多动障碍(ADHD),而在儿童晚期则表现出更多的 PTSD、品行/对立违抗性障碍和 ADHD。暴露于战争的儿童有更多的共病精神病理学,且共病数量随年龄增长而增加。值得注意的是,战争暴露使慢性病理学的患病率增加了 24 倍。包括母亲控制不住的风格和情绪困扰在内的母亲因素增加了早期和慢性精神病理学的风险,而减少儿童的社会参与则增加了晚期病理学的风险。
早期慢性应激不会自然愈合,其影响似乎随着时间的推移而加剧,随着年龄的增长,创伤暴露的儿童表现出更多的共病、慢性和外化特征。我们的研究结果表明,对创伤的反应是动态和可变的,并指出了母亲和儿童因素对风险和恢复轨迹的特定年龄影响。研究结果强调了进行长期随访研究以及在创伤暴露后制定个性化早期干预措施的重要性。